alpha-1 antitrypsin deficiency…

Medical experts recommend this detailed explanation.

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lungs, liver anatomy, aat deficiency, alpha-1 antitrypsin deficiency, lack of a liver protein, blocks the destructive effects of certain enzymes, lead to emphysema, liver disease, alpha-1 antitrypsin deficiency results, a genetic defect, mechanism that causes, associated liver disease, emphysema, people, deficiency is not known, however some evidence suggests that it related to inflammation, 75% of adults, severe deficiency, develop emphysema, 40 years of age, smoking, increase risk, shortness of breath, exertion, unintentional weight loss, wheezing, additional symptoms that associated, emphysema, cirrhosis, abnormal breathing pattern, exhalation takes more than twice, inspiration, agitation, ankle, feet, leg swelling, awakening from sleep not feeling rested, bloody, dark black, tarry bowel movements, melena, breast development in males, confusion, daytime sleepiness, difficulty falling asleep, staying asleep, insomnia, difficulty paying attention, dry mouth, excessive thirst, fatigue, fluctuating mood, headache, impaired concentration, impaired judgment, impotence, increased front-to-back diameter, the chest, barrel-shaped chest, irritability, poor temper control, light-headedness, fainting standing, memory loss, paleness, rapid heart rate, rising to a standing position, skin rash, lesion on the hands, feet, redness, sluggish, lethargic movement, swollen abdomen, increased girth, vision abnormalities, vomiting blood, vomiting material that looks, coffee grounds, a physical examination, reveal a barrel-shaped chest, listening, chest, a stethoscope, reveal wheezing, crackles, decreased breath sounds, following tests, diagnosis, arterial blood gases, serum alpha-1 antitrypsin level, chest x-ray, pulmonary function test, genetic testing, replacement, augmentation, therapy, the missing aat protein is available, not known how effective this is once disease has developed, people would benefit most, certainly, quitting smoking is crucial, treatments include bronchodilators, prompt antibiotics, upper respiratory tract infections, lung transplantation an option, who develop end-stage lung disease, genetic testing of relatives recommended, deficiency, not develop liver, lung disease, emphysema, cirrhosis, progressive diseases, kill, emphysema, cirrhosis, develop symptoms of alpha-1 antitrypsin deficiency.