 diabetes insipidus, nephrogenic…
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male urinary system, nephrogenic diabetes insipidus, acquired nephrogenic diabetes insipidus, nephrogenic diabetes insipidus, disorder characterized by the passage of large volumes of urine, due to a defect, the kidney tubules, diabetes insipidus-central, antidiuretic hormone, adh, vasopressin, hormone produced, in the hypothalamus, the brain, concentrates the urine by triggering the kidneys to reabsorb water, blood stream rather than excreting water, urine, nephrogenic diabetes insipidus, defect, in the kidney tubules, portion, the kidneys that causes, water to be excreted, reabsorbed, specific kidney defect, partial, complete failure of special receptors located on, the kidney tubules, adh, hormone that transmits the instruction to concentrate the urine, the cells, excessive amounts of water, excreted, the urine, producing a large quantity of very dilute urine, no response to vasopressin, though the blood level of this hormone is higher than normal, thirst mechanisms, adequate fluids, consumed, no significant effects on the fluid, electrolyte balance, inadequate fluids, consumed, high urine output, dehydration, high blood sodium, nephrogenic di, rare disorder, present at birth, a result of a sex-linked defect, congenital nephrogenic di, affects men, women, pass the gene on to children, most commonly, nephrogenic diabetes insipidus, acquired disorder, precipitating factors include drugs, lithium, demeclocycline, amphotericin b, electrolyte disorders, high calcium, low potassium levels, urinary obstruction, excessive thirst, intense, uncontrollable, a craving, ice water, excessive urine volume, exceed 3 to 15 liters per day, inadequate fluid consumption, result in, dehydration, dry skin, dry mucous membranes, sunken appearance to eyes, sunken fontanelles, soft spot, in infants, fatigue, lethargy, headache, irritability, low body temperature, muscle pains, rapid heart rate, weight loss, examination, indicate dehydration, shock, fluid intake is inadequate, pulse rate rapid, a low blood pressure present, most significant indication of diabetes insipidus is persistent high urine output regardless of fluid intake, signs associated, high urine output, high serum osmolality, low urine osmolality, high adh levels, kidneys not making a more concentrated urine, the person is given adh, tests, urine specific gravity, urine concentration test, urine 24h volume, serum sodium, goal of treatment, regulate fluid levels, all cases, consistently high fluid intake, volume of fluids consumed should approximately equal the volume of urine produced, reduction, discontinuation of medications, nephrogenic di, improve symptoms, hydrochlorothiazide, improve symptoms, used alone, in combination, medications, indomethacin, this medication, diuretic, medications, increase urine output, hydrochlorothiazide, reduce the urine output, nephrogenic di, medication works by causing sodium, water to be excreted, in the early part, the renal tubules, proximal tubules, leaves less fluid available, the late portion, the kidney, distal tubule, to excrete, the portion, nephrogenic di, limits the total volume of urine, excreted, congenital nephrogenic di, chronic condition requiring lifelong treatment, acquired nephrogenic di short-term, long-term, severe dehydration, shock, inadequate fluid intake, hypernatremia, high blood sodium, dilation, the ureters, bladder, symptoms indicate diabetes insipidus present, no known prevention, congenital nephrogenic diabetes insipidus, treatment of causative disorders, prevent some cases of acquired nephrogenic di, medications should only be used under the supervision, the health care provider.
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